Though antineoplastic drugs have demonstrated benefits as a cancer treatment, they pose significant occupational health risks to clinicians administering the drugs. The Centers for Disease Control and ...

Cancer cells frequently develop the ability to expel anticancer drugs before they can work—a phenomenon called multidrug resistance (MDR)—which is one of the leading reasons why chemotherapy fails in ...

Selection of Germline Genetic Testing Panels in Patients With Cancer: ASCO Guideline Clinical Insights ASCO Standards are evidence-based standards to provide frameworks for best practices in cancer ...

Cancer cells frequently develop the ability to expel anticancer drugs before they can work -- a phenomenon called multidrug ...

Research by UMass Chan Medical School scientists Sharon Cantor, Ph.D., and Jenna M. Whalen, Ph.D., poses a new explanation for how cancer-fighting drugs attack and destroy BRCA1 and BRCA2 tumor cells.

ASCO Guidelines provide recommendations with comprehensive review and analyses of the relevant literature for each recommendation, following the guideline development process as outlined in the ASCO ...

An emerging class of anticancer drugs called EZH2 inhibitors may greatly enhance the potency of some cancer immunotherapies. An emerging class of anticancer drugs called EZH2 inhibitors may greatly ...

Eli Lilly’s obesity and diabetes treatment ‘Mounjaro’ has overtaken MSD, Merck in the US’s anticancer drug ‘Keytruda’ to ...

Oscotec forms platform team to advance resistance-proof cancer therapies in Korea New platform unit, led by Pasteur veteran ...

BRCA1 and BRCA2 genes play a crucial role in repairing damaged DNA. Mutations in BRCA1 and BRCA2 substantially increase the likelihood of breast and ovarian cancer, respectively. This new research ...

In a recent study published in the journal Scientific Reports, researchers leveraged the knowledge that cancers and specific types of dementia share pathophysiological underpinnings to investigate the ...

Senescent cells walk a tightrope, risking cell death with high levels of iron and other damaging agents, but compensating for this by overproducing a protective protein, GPX4, which staves off death.